Community Shares | October 28th 2024
This Week’s Articles:
- Researchers are investigating how environmental factors, such as exposure to pyrethroid insecticides, may interact with genetic predispositions to increase the risk of ASD.
- A recent meta-analysis highlights that executive function delays—such as difficulties with attention, impulse control, switching focus, planning, organizing, and problem-solving—are common across neurodevelopmental conditions, including ADHD, autism and Tourette’s syndrome.
- A new study has examined the link between prenatal exposure to particulate matter (PM2.5) from specific sources and the risk of ASD in over 300,000 mother-child pairs in southern California.
- Don’t miss this opportunity to explore groundbreaking research on the effects of prenatal exposure to bisphenol A (BPA), a common endocrine-disrupting chemical found in plastics, and its potential role in neurodevelopmental disorders like ASD.
Children With Autism and ADHD Shown to Struggle to Detoxify Common Plastic Additive
A recent study conducted by Rowan University and Rutgers University revealed significant differences in how children with autism spectrum disorder (ASD) and attention deficit hyperactivity disorder (ADHD) metabolize bisphenol A (BPA), a common plastic additive, compared to neurotypical children.
New Australian Study Links BPA Exposure to Higher Autism Risk in Males
A recent study conducted in Australia and published in Nature Communications reveals a concerning link between bisphenol A (BPA), a common plastic chemical, and an increased risk of autism in boys. Researchers from Florey Institute found that mothers with higher BPA levels in their urine during pregnancy had sons who were 3.5 times more likely to show autism symptoms by age 2 and 6 times more likely to receive an autism diagnosis by age 11.
SafeMinds Research for Autism Health
A study in Environmental Health Perspectives by Emily C. Somers and colleagues found that women of childbearing age with higher mercury levels in hair or blood were at much higher risk of developing antinuclear antibodies, a marker of autoimmunity.
